Je compte acqu?rir une Tablette GalaxyTab 10.1.
Je voudrais savoir si la tablette dispose d'un logiciel de lecture de vid?o (id?alement VLC) afin de pouvoir regarder la TV en WIFI avec le service Multiposte de Free.
En vous remerciant d'avance je vous souhaite une bonne soir?e
Source: http://forum.frandroid.com/topic/86054-galaxy-tab-101-et-multiposte-free/
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WASHINGTON ? A politically connected financial firm and its founder pleaded guilty Friday to taking part in fraud and bid-rigging conspiracies related to the municipal bond business.
The Justice Department said CDR Financial Products Inc. of Beverly Hills, Calif., and owner David Rubin entered guilty pleas in federal court in Manhattan. The department said they acknowledged their roles in schemes designed to win contracts to invest the proceeds of municipal bonds issued by state, county and local governments.
Rubin faces up to 20 years in prison.
Two other CDR executives are scheduled to go on trial in New York next week.
CDR also was investigated for its ties to former New Mexico Gov. Bill Richardson and business the company won in that state. No charges grew out of that investigation.
Rubin is the tenth person to plead guilty to criminal charges in the ongoing federal investigation of the municipal bonds industry, the department said. Eight others have been charged, including the two CDR executives.
The government so far has recovered $743 million in restitution and penalties from five banks, Bank of America Corp., UBS AG, JPMorgan Chase and Co., GE Funding Capital Market Services Inc. and Wachovia Bank, since acquired by Wells Fargo and Co.
Municipal bonds are issued to build schools, hospitals and roads in a $2.8 trillion market. About $100 billion of the proceeds from the bond sales each year are temporarily invested before being used for the original purpose of the sales.
The government launched a widespread investigation into the business of reinvesting municipal bond proceeds after Bank of America came forward and disclosed to authorities that its investment division paid for information that helped the bank gain an advantage with local governments that were looking to invest their proceeds from municipal bond sales. Because nearly all the municipal bonds involved were tax-exempt, they are regulated by the Internal Revenue Service.
In the CDR case, Rubin admitted that over eight years beginning in 1998, he provided information to financial institutions and insurance companies that helped win bids, intentionally solicited losing bids and also paid kickbacks in what was supposed to be a competitive situation, Justice said.
Rubin's guilty plea follows his failed effort to delay his trial because of his wife's worsening medical condition. In court papers, Rubin said Gail Rubin is in the final stages of a long fight with pancreatic cancer and asked to put off his trial until 35 days after her death. The Rubins have been married 26 years and have seven children, according to court papers.
U.S. District Judge Victor Marrero denied the request, a decision upheld by a panel of federal appellate judges in New York. Rubin and CDR, also known as Rubin/Chambers, Dunhill Insurance Services Inc., were first indicted in 2009.
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Infografik: Farewell To A Genius - Steve Jobs | BENM.AT i.benm.at/w3W8pA 
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Source: http://twitter.com/bensen/statuses/152709109429440512
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DES MOINES, Iowa ? Republican presidential hopeful Newt Gingrich once praised the health care law enacted in Massachusetts by then-Gov. Mitt Romney.
In an April 2006 memo, the former House speaker called it "the most exciting development of the past few weeks." Gingrich also said the law has "tremendous potential to effect major change in the American health system."
The memo from Gingrich's Atlanta-based Center for Health Transformation came to light Tuesday as the GOP candidate set out on a 22-stop bus tour of Iowa in the run-up to the state's Jan. 3 caucuses.
The memo also noted shortcomings in the Massachusetts law. Gingrich said the state's many regulations prohibited insurers from offering cheaper plans that would make coverage affordable.
But Gingrich went on to note that that "we agree entirely with Gov. Romney and Massachusetts legislators that our goal should be 100 percent insurance coverage for all Americans."
Gingrich and others have argued that the Massachusetts law, widely seen as the model for President Barack Obama's national health overhaul, undercuts Romney's conservative credentials.
Gingrich also has faced questions about his past support for an individual health care mandate, anathema to conservatives.
A Gingrich spokesman said the former Georgia congressman's comments are "old news that has been covered already."
"Newt previously supported a mandate for health insurance and changed his mind after seeing its effects," said the spokesman, R.C. Hammond. "The real question is why `Mitt the Massachusetts Moderate' won't admit that health insurance mandates don't work."
Gingrich and Romney have sparred over which one of them is truly conservative.
The Gingrich memo was first reported Tuesday by The Wall Street Journal.
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SAN ANTONIO -- Since winning the Heisman Trophy, Robert Griffin III is often shadowed by a bodyguard of sorts to dissuade autograph seekers, and this week tucked his dreadlocks under a hat in hopes of strolling through Sea World incognito.
"I got a lot of double-takes," the Baylor quarterback said. "If you can get a double-take, you can walk far enough away to where they'll be discouraged to approach you. But it was cool. I didn't mind."
Now it's a question of whether RG3 is about to give college football its last look at him altogether.
The nation's most electrifying player leads No. 15 Baylor (9-3) into the Alamo Bowl against Washington (7-5) on Thursday night while keeping his decision about leaving for the NFL private for now.
Griffin, who says he can't "go to Wendy's and get a cheeseburger without signing 1,000 autographs" since winning college football's top award, reiterated in San Antonio that he's undecided about forgoing his senior year. He said his parents are looking at his draft prospects but denies having any substantial talks with them.
Baylor can hardly feel jilted if this is Griffin's last game.
The fourth-year junior, who also won the Davey O'Brien Award and is the AP Player of the Year, has raised the program's profile to unseen heights. He rescued the Bears from their perennial status as the Big 12's punch line and has Baylor on a five-game winning streak, its longest in 20 years.
A win against Washington would match the school record of 10 wins when Mike Singletary was a senior in 1980, and merely playing in back-to-back bowls is a first for Baylor in two decades. Simply put, it's been a magical season the school doesn't want to see end.
Washington won't exactly say the same.
The Huskies stumbled into a second consecutive bowl game dropping four of their last six and losing badly to all four ranked teams they played this season. That included Stanford and Andrew Luck, the Heisman runner-up to Griffin, who coasted in a 65-21 win that began Washington's second-half slide.
Yet tailback Chris Polk and other seniors still vividly remember going 0-12 just four years ago under Tyrone Willingham. According to the school, Washington is the first BCS program to go from winless to back-to-back bowl appearances in three years since Central Florida in 2004.
"I would have never imagined this," offensive lineman Senio Kelemte said. "It was pretty hard for all of us, the 0-12 season. I'm pretty sure a lot of guys didn't really want to play football anymore or wanted to transfer or just ... just football wasn't fun."
The Huskies have a shot at an eight-win season for the first time since 2001, but it might be a long night against Baylor.
The Huskies will put one of the nation's worst defenses against the Bears, whose offense was the second-best in the country. Baylor averaged more than 570 yards of offense a game behind Griffin, who threw for nearly for 3,998 yards with a Big 12-leading 36 touchdowns and only six interceptions. That made him the nation's most efficient passer.
Baylor averaged 43 points a game. Washington's let opponents score an average of 33.
"We've had a huge challenge this whole year playing against good offenses," Washington defensive coordinator Nick Holt said. "This is good offense and the only difference this time is that we're playing against the best player in the country and a Heisman Trophy winner who has a great supporting cast."
Anything else?
"And, oh yeah," Holt added. "They run an up-tempo, no huddle offense and can score really quickly."
Griffin is the first Heisman winner to play in a bowl game before New Year's Day since Ty Detmer led BYU to the Holiday Bowl in 1990. Two years later, Baylor won its last postseason game in the Sun Bowl.
Ending that drought may be the last thing left for Griffin for do.
"We know why we're here and we came to win our 10th game," Griffin said. "Washington just happens to be in the way."
Source: http://feedproxy.google.com/~r/cbssportsline/home_news/~3/exfaBuDlwgM/rss
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MADISON, Wis.?? The Green Bay Packers have an MVP candidate in quarterback Aaron Rodgers, a Super Bowl championship won just 10 months ago and an undefeated team making a run toward another title for Titletown.
The Packers now have hundreds of new owners, too.
The team kicked off a rare stock sale Tuesday to help pay for another round of renovations at Lambeau Field, giving pretty much anyone a shot at becoming an NFL owner for $250 per share, plus a $25 handling charge.
Sarah Johnson, 34, of Portage, said it took her nearly 20 minutes to complete what should have been a 30-second process, but it was worth to wait.
"I could have just as well thrown my money out the window for what I get for it, other than a feel-good," she said. "I just feel like the Packer organization has sort of a nostalgia and an excitement around it other franchises don't have. Just to say you're part of that on some level is neat to me."
The team received 1,600 orders in the first 11 minutes of the sale, said Packers President Mark Murphy, who had to reassure fans the Packers website was still working. Team spokesman Aaron Popkey said he did not have any sales data as of late Tuesday evening.
"It's just a question of volume," Murphy said. "Fans are excited about this opportunity. We just encourage fans to be patient."
It is the fifth stock sale in the Packers' 92-year history and the first in 14 years.
The NFL's only publicly-owned team offered 250,000 shares through Feb. 29, subject to an extension. The stock isn't an investment in the traditional sense: Its value doesn't increase, there are no dividends, it has virtually no re-sale value and it won't give buyers a leg up on the 93,000 people on the waiting list for season tickets.
The fine print on the Packers' stock sale website says, too, that NFL rules prohibit shareholders from betting on any NFL game; violators could face up to $5,000 in fines.
Popkey and NFL spokesman Greg Aiello said in emails to The Associated Press that shareholder betting has never been a problem in the past.
What buyers do get is a piece of paper declaring them a team owner, voting rights and the right to attend the annual stockholder meeting at Lambeau each summer before training camp. Oh, and they get access to a special line of shareholder apparel, too.
The economy is still lurching along, but that probably won't make much difference to the cheesehead nation, among the league's most dedicated fans. The Packers' timing is perfect, too. Christmas is only a few weeks off and the Packers are hot, hot, hot: The defending champions clinched the NFC North title this past weekend and look like a favorite again with a 12-0 record with four games left to play in the regular season.
Plus, the cause is nothing less than spiffing up the team's hallowed frozen tundra.
The team hopes to generate at least $22 million through the stock sale to help defray the cost of a $143 million renovation project at Lambeau. Plans call for adding 6,700 additional seats, new high-definition video screens and a new entrance by 2013.
Before the sale, there were 112,205 Packers stockholders who own a total of 4.75 million shares. The latest sale did have restrictions: Stock can only be purchased by individuals, not businesses, and there's a 200-share cap, a figure that includes any stock purchased during the last sale in 1997.
The offering is limited to people with addresses in the U.S., Guam, Puerto Rico and the U.S. Virgin Islands. Soldiers and U.S. residents who are currently overseas have to use their U.S. addresses.
The Packers have been a publicly owned nonprofit corporation since 1923. The team held its first stock sale that year, followed by sales in 1935 and 1950 that helped keep the franchise afloat even as other small-markets teams were sinking.
Back in 1997, the last time the Packers offered stock, then-team president Bob Harlan was looking for ways to cover stadium renovation costs. He recalled that other owners balked, worried that the Packers would use the money to compensate their coaches or improve their roster in a way other teams couldn't. It was only after Commissioner Paul Tagliabue and Pittsburgh Steelers owner Dan Rooney argued in favor of the idea that the proposal passed. Rooney said the Packers deserved unanimous support because they were a vital part of NFL history.
Some 400,000 shares went on sale that year for $200 apiece. About 120,000 were sold, raising $24 million.
Ryan Vaubel, 35, a lifelong Packer fan from Sun Prairie, Wis., said he can't justify spending $275 on a piece of paper. But he understands the Packers need money; he still remembers when the team sold chunks of Lambeau sod following the team's 1997 Super Bowl victory.
"I'm not personally disappointed. All you can say is you have a piece of paper," Vaubel said. "It's more or less just a way to donate money to the team so they can raise more revenue and remain competitive on the field."
Nicole Kappus Solheid, 37, of Apple Valley, Minn., grew up in Chippewa Falls, Wis. She said she was strongly considering buying a share. For her, it's a matter of pride; her husband is a Viking fan.
"I'm still from Wisconsin and I'm still very proud of where I come from," she said. "I'd be very proud to put that stock option up on my wall and show my neighbors. It's just to be a part of (the Packers) legacy."
Copyright 2011 The Associated Press. All rights reserved. This material may not be published, broadcast, rewritten or redistributed.
Source: http://www.msnbc.msn.com/id/45567251/ns/business-stocks_and_economy/
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In October, Kendall Goheen of Richmond British Columbia allegedly stabbed and killed the pets that he co-owned with his ex-wife. He has been charged with nine counts of animal cruelty.
I published this because (to date) this man is reportedly still not under arrest. This story needs to remain in the news.
The family's 70-pound boxer and two cats suffered multiple stab wounds and other trauma, before being placed in a freezer in Goheen's home.
His ex-wife, Latia Del Riviero, who found the animals in the freezer Oct. 7. She called police.
The local SPCA was called to the house but when officers arrived, Goheen refused to speak to them. Goheen allegedly left the animals' bodies in a plastic container outside his house. No wonder, officials with the BC SPCA called it one of the most disturbing cases they have encountered in recent memory, according to a news account, CBC Canada.
Goheen's charges fall under both the B.C. Prevention of Cruelty to Animals Act and the Criminal Code of Canada.
According to another news account, Del Riviero said Goheen loved the animals, but blamed the medication he had been taking for depression.? Apparently, he had recently lost his job.
Goheen is now believed to be in Saskatchewan; a warrant has been issued for his arrest. However, to date, he reportedly remains free. What's up with that
There's lots of data to indicate that when people are readily able to commit these sorts of heinous crimes to animals, it's only a matter of people before people are also attacked. Blame the drugs or not, at some point we need to be responsible for our own actions.
?
Filed under: animal cruelty, animals, cats, dogs
Tags: animal cruelty, BC SPCA, heinous crmes to animals, Kendall Goheen, killing pets, murdering anmals, stabbing pets, Steve Dale archives
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Fans tear down a goal post, injuring some participants, after Oklahoma State defeated Oklahoma 44-10 in an NCAA college football game in Stillwater, Okla., Saturday, Dec. 3, 2011. (AP Photo/Brody Schmidt)
Fans tear down a goal post, injuring some participants, after Oklahoma State defeated Oklahoma 44-10 in an NCAA college football game in Stillwater, Okla., Saturday, Dec. 3, 2011. (AP Photo/Brody Schmidt)
Oklahoma State quarterback Brandon Weeden, center, celebrates with fans following a 44-10 victory over Oklahoma in an NCAA college football game in Stillwater, Okla., Saturday, Dec. 3, 2011. (AP Photo/Sue Ogrocki)
A fan hangs from the goal post it was tore down in celebration of Oklahoma State's 44-10 win over Oklahoma in an NCAA college football game in Stillwater, Okla., Saturday, Dec. 3, 2011. (AP Photo/Sue Ogrocki)
Oklahoma State fans carry a goal post they tore down following the Cowboy's 44-10 win of rival Oklahoma in an NCAA college football game in Stillwater, Okla., Saturday, Dec. 3, 2011. (AP Photo/Brody Schmidt)
Oklahoma State quarterback Brandon Weeden, center, celebrates with fans following a 44-10 victory over Oklahoma in an NCAA college football game in Stillwater, Okla., Saturday, Dec. 3, 2011. (AP Photo/Sue Ogrocki)
STILLWATER, Okla. (AP) ? Thousands of fans stormed the field and tore down goalposts after Oklahoma State's 44-10 victory over archrival Oklahoma, leaving at least 13 people injured, including two in critical condition, an emergency medical official said early Sunday.
Michael Authement, who heads the command post at emergency medical provider LifeNet EMS, told The Associated Press that a throng so big took to the field as the game ended that some fans were trampled and one person fell at least 15 feet onto concrete during a wild celebration by Oklahoma State fans.
"They won the game and stormed the field and ripped down the goalposts and some were jumping off the stands and hit the field and others got trampled. It was a nasty deal," Authement said.
He said the crowd was so big it took police at least 45 minutes to clear the crowd from the field at Boone Pickens Stadium in Stillwater.
"There were thousands of people. Thousands of people stormed the field. You couldn't move there were so many people," he added.
Authement said nine ambulances, including six from LifeNet, rushed 11 of the injured away and the two in critical condition were flown to Oklahoma City hospitals. He said he knew of leg fractures but didn't have any details on the extent of the injuries, though two of the 13 had minor injuries and were treated at the scene and released.
An Oklahoma State University police central dispatcher said she had no immediate details to release when contacted by AP and the public information officer did not immediately return messages seeking comment. Stillwater police and the Oklahoma highway patrol also had no immediate comment.
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The appearance of oxygen in the Earth's atmosphere probably did not occur as a single event, but as a long series of starts and stops, according to an international team of researchers who investigated rock cores from the FAR DEEP project.
The Fennoscandia Arctic Russia - Drilling Early Earth Project -- FAR DEEP -- took place during the summer of 2007 near Murmansk in the northwest region of Russia. The project, part of the International Continental Scientific Drilling Program, drilled a series of shallow, two-inch diameter cores and, by overlapping them, created a record representing stone deposited during the Proterozoic Eon -- 2,500 million to 542 million years ago.
"We've always thought that oxygen came into the atmosphere really quickly during an event," said Lee R. Kump, professor and head of geosciences, Penn State. "We are no longer looking for an event. Now we are looking for when and why oxygen became a stable part of the Earth's atmosphere."
The researchers report in today's (Dec. 1) issue of ScienceExpress that evaluation of these cores and comparison with cores from Gabon previously analyzed by others, supports the conclusion that the Great Oxidation Event played out over hundreds of millions of years. Oxygen levels gradually crossed the low atmospheric oxygen threshold for pyrite -- an iron sulfur mineral -- oxidation by 2,500 million years ago and the loss of any mass-independently fractionated sulfur by 2,400 million years ago. Then oxygen levels rose at an ever-increasing rate through the Paleoproterozoic, achieving about 1 percent of the present atmospheric level.
"The definition of when an oxygen atmosphere occurred depends on which threshold you are looking for," said Kump. "It could be when pyrite becomes oxidized, when sulfur MIF disappears or when deep crustal oxidation occurs."
When the mass-independent fractionated sulfur disappeared, the air on Earth was still not breathable by animal standards. When red rocks containing iron oxides appeared 2,300 million years ago, the air was still unbreathable.
"At about 1 percent oxygen, the groundwater became strongly oxidized, making it possible for groundwater seeping through rocks to oxidize organic materials," said Kump.
Initially, any oxygen in the atmosphere, produced by the photosynthesis of single-celled organisms, was used up when sulfur, iron and other elements oxidized. When sufficient oxygen accumulated in the atmosphere, it permeated the groundwater and began oxidizing buried organic material, oxidizing carbon to create carbon dioxide.
The cores from the FAR-DEEP project were compared with the Francevillian samples from Gabon using the ratio of carbon isotopes 13 and 12 to see if the evidence for high rates of oxygen accumulation existed worldwide. Both the FAR-DEEP project's cores and the Francevillian cores show large deposits of carbon in the form of fossilized petroleum. Both sets of cores also show similar changes in carbon 13 through time, indicating that the changes in carbon isotopes occurred worldwide and oxygen levels throughout the atmosphere were high.
"Although others have documented huge carbon isotope variations at later times in Earth history associated with stepwise increases in atmospheric oxygen, our results are less equivocal because we have many lines of data all pointing to the same thing," said Kump. "These indications include not only carbon13 isotope profiles in organic mater from two widely separated locations, but also supporting profiles in limestones and no indication that processes occurring since that time have altered the signal."
###
Penn State: http://live.psu.edu
Thanks to Penn State for this article.
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Source: http://www.labspaces.net/115656/Rise_of_atmospheric_oxygen_more_complicated_than_previously_thought
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ScienceDaily (Dec. 1, 2011) ? Shortening end caps on chromosomes in human cervical cancer cells disrupts DNA repair signaling, increases the cells' sensitivity to radiation treatment and kills them more quickly, according to a study in Cancer Prevention Research.
Researchers would to like see their laboratory findings -- published in the journal's Dec. 5 print edition -- lead to safer, more effective combination therapies for hard-to-treat pediatric brain cancers like medulloblastoma and high-grade gliomas. To this end, they are starting laboratory tests on brain cancer cells.
"Children with pediatric brain cancers don't have very many options because progress to find new treatments has been limited the last 30 years," said Rachid Drissi, PhD, principal investigator on the study and a researcher in the Division of Oncology at Cincinnati Children's. "The ability to make cancer cells more sensitive to radiation could allow physicians to use lower radiation doses to lessen side effects. Too many children with brain cancer can develop disabilities or die from treatment."
Before treating cells with ionizing radiation, the researchers blocked an enzyme called telomerase, found in over 90 percent of cancer cells but barely detectable in most normal human cells. In cancer cells, telomerase helps maintain the length of caps on the ends of chromosomes called telomeres. This helps cancer cells replicate indefinitely, grow and spread, Drissi said.
Unraveling DNA stability
Found on chromosomes in both cancerous and normal cells, telomeres are analogous to plastic caps that keep shoestring ends from unraveling. Telomeres help preserve DNA stability in cells by containing genetic miscues. This helps explain why cells with maintained or long telomeres appear to be more resistant to radiation.
In normal cells lacking the telomerase enzyme, telomeres get shorter each time cells divide. They continue doing so until normal cells stop dividing, reaching a condition called senescence. If this first cell-cycle "stop sign" is bypassed, cells continue dividing until telomeres become critically short and reach a second stopping point, when most cells die. In rare instances, cells bypass this second "stop sign" and survive. This survival is often associated with telomerase activation and the onset of cancer.
This was the basis for experiments Drissi and his colleagues conducted to compare the radiation sensitivity and survivability of cells based on telomere length. They also monitored DNA repair responses in the cells by looking for specific biochemical signs that indicate whether the repair systems are working.
The tests involved normal human foreskin cells -- called fibroblasts -- and human cervical carcinoma cells. They exposed the cells to ionizing radiation and analyzed DNA repair responses as telomeres became progressively shorter. In the cervical cancer cells, researchers blocked the telomerase enzyme before radiation treatment to induce progressively shorter telomeres.
Both late-stage noncancerous cells with shorter telomeres, and cancer cells with induced shorter telomeres, were more radiosensitive and died more quickly, according to the study.
Among cancer cells with maintained telomere length, close to 10 percent receiving the maximum dose of ionizing radiation used in the study (8 Gy, or Gray Units) survived the treatment. None of the cancer cells with the shortest telomeres survived that exposure.
Researchers said the cancer cells became more radiosensitive because material inside the chromosomes -- called chromatin -- compacted as telomeres became shorter. Compacted chromatin then disrupted the biochemical signaling of a protein called ATM (ataxiatelangeietasia mutated).
ATM is a master regulator of DNA repair and cell division. It sends signals to activate other biochemical targets (H2AX, SMC1, NBS1 and p53) that help direct DNA repair and preserve genetic stability. In telomere-shortened cancer cells, the compacted chromatin inhibited ATM signaling to all of the chromatin-bound targets tested in the study. This disrupted DNA repair responses and increased radiation sensitivity.
Testing brain cancer cells
The researchers are now testing their findings in cells from hard-to-treat pediatric brain tumors. These tests begin as Drissi's laboratory also leads correlative cancer biology studies of tumor samples from a current clinical trial. The trial is evaluating telomere shortening as a stand-alone therapy for pediatric cancer.
Managed through the National Institutes of Health's Children's Oncology Group (COG), the multi-institutional Phase 1 trial is testing the safety and tumor response capabilities of the drug Imetelstat, which blocks telomerase in cancer cells. Drissi serves on the clinical trial committee along with Maryam Fouladi, MD, MSc, and medical director of Neuro-Oncology at Cincinnati Children's. She leads the medical center's clinical participation in the trial.
Drissi and Fouladi are starting preparatory work to develop, and seek approvals for, a possible clinical trial to test telomere shortening and radiation treatment as a safer, more effective treatment for pediatric brain tumors.
Funding support for the current study in Cancer Prevention Research -- published by the American Society for Cancer Research -- came from the National Institutes of Health, the American Lebanese Syrian Associated Charities of St. Jude Children's Research Hospital and Cincinnati Children's Hospital Medical Center. Also collaborating were researchers from Children's National Medical Center in Washington, D.C., and from St. Jude. Funding support for the Drissi lab's correlative studies on the COG clinical trial comes from CancerFree Kids Pediatric Cancer Research Alliance and from Children's Cancer Research Fund.
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Source: http://www.sciencedaily.com/releases/2011/12/111201105441.htm
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Continue reading LG updates eye-tracking, glasses-free 3D displays, learns to love the hyphen
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We know, we know. Your pet is the cutest. Perhaps this contest should simply be to find the second cutest pet of 2011, since we already know that your pet is #1. In reality though, this is less a competition and more a celebration of all the great pets who brighten our days.
Over the past year, pets have been adored around the world. There's Boo the Dog with his fluffy head and mini-sweaters, the pug who hates the iPhone, the labrador puppies running in slow motion, the dog with the best smile ever and the irresistible litter of 16 Dalmatian pups.
Cats weren't to be upstaged this year, from the one obsessed with bubble wrap, to the blind kitten playing with his first toys, to the cat taking on an iPad.
Unfortunately, not all pets are as loved as the ones basking in viral video glory. Millions of stray dogs and cats are euthanized each year in the U.S. If you have pets, be sure to spay/neuter them. If you are thinking about getting a pet, consider the long-term responsibility of pet ownership, and explore adoption options with your local shelter, or by visiting Petfinder.com and the ASPCA website.
Check out the photos below to vote for the cutest pet of 2011, and don't forget to submit your own pets!
(Note: Photos have to be approved before they will appear in the slideshow. Please limit to one submission per pet.)
Add a slide
Click 'Add Slide,' upload an image of your pet, add its name for the title, and describe in the caption what makes your animal the cutest!
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'; var coords = [-5, -72]; // display fb-bubble FloatingPrompt.embed(this, html, undefined, 'top', {fp_intersects:1, timeout_remove:2000,ignore_arrow: true, width:236, add_xy:coords, class_name: 'clear-overlay'}); });Source: http://www.huffingtonpost.com/2011/12/01/cutest-pets-of-2011-submi_n_1119188.html
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schizophrenia or prophecy? this is a hard film to score. its at least a 4 star film, and potentially 5, so ill go in between. it doesnt have a high re-watchability factor which is my only criticism, and that isnt really a criticism because its more a product of the material then the quality of the film. this is easily one of the best films of the year, and one of the better films in the past few years. shannon and chastain were both amazing in their roles, which for chastain amazes me because she has so little experience in film, and even the supporting players around them all added wonderful performances. the subject matter is as engaging as it gets, and as i hoped for through the entire film, things are not what they seem. beautiful direction by nichols, amazing cinematography for such a small scale story, and the material was handled with perfect care. nichols, shannon, and chastain probably all deserve oscar noms, and this is the type of soul stirring film that must be seen. i havent felt like this leaving a film since "a serious man".
November 7, 2011Source: http://www.rottentomatoes.com/m/take_shelter/
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